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Animals
S. Eketjäll, Janson, J., Kaspersson, K., Bogstedt, A., Jeppsson, F., Fälting, J., Haeberlein, S. Budd, Kugler, A. R., Alexander, R. C., and Cebers, G., “AZD3293: A Novel, Orally Active BACE1 Inhibitor with High Potency and Permeability and Markedly Slow Off-Rate Kinetics.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1109-23, 2016.
K. Krishna, Behnisch, T., and Sajikumar, S., “Inhibition of Histone Deacetylase 3 Restores Amyloid-β Oligomer-Induced Plasticity Deficit in Hippocampal CA1 Pyramidal Neurons.”, J Alzheimers Dis, vol. 51, no. 3, pp. 783-91, 2016.
C. Peters, Bascuñán, D., Opazo, C., and Aguayo, L. G., “Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.”, J Alzheimers Dis, vol. 51, no. 3, pp. 689-99, 2016.
E. Aso, Andrés-Benito, P., Carmona, M., Maldonado, R., and Ferrer, I., “Cannabinoid Receptor 2 Participates in Amyloid-β Processing in a Mouse Model of Alzheimer's Disease but Plays a Minor Role in the Therapeutic Properties of a Cannabis-Based Medicine.”, J Alzheimers Dis, vol. 51, no. 2, pp. 489-500, 2016.
A. S. R. Sierksma, de Nijs, L., Hoogland, G., Vanmierlo, T., van Leeuwen, F. W., Rutten, B. P. F., Steinbusch, H. W. M., Prickaerts, J., and van den Hove, D. L. A., “Fluoxetine Treatment Induces Seizure Behavior and Premature Death in APPswe/PS1dE9 Mice.”, J Alzheimers Dis, vol. 51, no. 3, pp. 677-82, 2016.
K. A. Southam, Vincent, A. J., and Small, D. H., “Do Microglia Default on Network Maintenance in Alzheimer's Disease?”, J Alzheimers Dis, vol. 51, no. 3, pp. 657-69, 2016.
R. Pluta, Kocki, J., Ułamek-Kozioł, M., Petniak, A., Gil-Kulik, P., Januszewski, S., Bogucki, J., Jabłoński, M., Brzozowska, J., Furmaga-Jabłońska, W., Bogucka-Kocka, A., and Czuczwar, S. J., “Discrepancy in Expression of β-Secretase and Amyloid-β Protein Precursor in Alzheimer-Related Genes in the Rat Medial Temporal Lobe Cortex Following Transient Global Brain Ischemia.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1023-31, 2016.
G. Lou, Zhang, Q., Xiao, F., Xiang, Q., Su, Z., and Huang, Y., “Intranasal TAT-haFGF Improves Cognition and Amyloid-β Pathology in an AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 985-90, 2016.
S. K. Singhrao, Harding, A., Chukkapalli, S., Olsen, I., Kesavalu, L., and Crean, S. J., “Apolipoprotein E Related Co-Morbidities and Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 935-48, 2016.
M. Elahi, Hasan, Z., Motoi, Y., Matsumoto, S. - E., Ishiguro, K., and Hattori, N., “Region-Specific Vulnerability to Oxidative Stress, Neuroinflammation, and Tau Hyperphosphorylation in Experimental Diabetes Mellitus Mice.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1209-24, 2016.
B. Ongali, Nicolakakis, N., Tong, X. - K., Aboulkassim, T., Imboden, H., and Hamel, E., “Enalapril Alone or Co-Administered with Losartan Rescues Cerebrovascular Dysfunction, but not Mnemonic Deficits or Amyloidosis in a Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1183-95, 2016.
F. Guillot, Kemppainen, S., Lavasseur, G., Miettinen, P. O., Laroche, S., Tanila, H., and Davis, S., “Brain-Specific Basal and Novelty-Induced Alternations in PI3K-Akt and MAPK/ERK Signaling in a Middle-Aged AβPP/PS1 Mouse Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1157-73, 2016.
Y. Kim, Kim, C., Jang, H. Young, and Mook-Jung, I., “Inhibition of Cholesterol Biosynthesis Reduces γ-Secretase Activity and Amyloid-β Generation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1057-68, 2016.
P. Adam, Křížková, S., Heger, Z., Babula, P., Pekařík, V., Vaculovičoá, M., Gomes, C. M., Kizek, R., and Adam, V., “Metallothioneins in Prion- and Amyloid-Related Diseases.”, J Alzheimers Dis, vol. 51, no. 3, pp. 637-56, 2016.
A. M. Masoud, Bihaqi, S. W., Machan, J. T., Zawia, N. H., and Renehan, W. E., “Early-Life Exposure to Lead (Pb) Alters the Expression of microRNA that Target Proteins Associated with Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1257-64, 2016.
N. Lelental, Brandner, S., Kofanova, O., Blennow, K., Zetterberg, H., Andreasson, U., Engelborghs, S., Mroczko, B., Gabryelewicz, T., Teunissen, C., Mollenhauer, B., Parnetti, L., Chiasserini, D., Molinuevo, J. Luis, Perret-Liaudet, A., Verbeek, M. M., Andreasen, N., Brosseron, F., Bahl, J. M. C., Herukka, S. - K., Hausner, L., Frölich, L., Labonte, A., Poirier, J., Miller, A. - M., Zilka, N., Kovacech, B., Urbani, A., Suardi, S., Oliveira, C., Baldeiras, I., Dubois, B., Rot, U., Lehmann, S., Skinningsrud, A., Betsou, F., Wiltfang, J., Gkatzima, O., Winblad, B., Buchfelder, M., Kornhuber, J., and Lewczuk, P., “Comparison of Different Matrices as Potential Quality Control Samples for Neurochemical Dementia Diagnostics.”, J Alzheimers Dis, vol. 52, no. 1, pp. 51-64, 2016.
A. Tramutola, Pupo, G., Di Domenico, F., Barone, E., Arena, A., Lanzillotta, C., Broekaart, D., Blarzino, C., Head, E., D Butterfield, A., and Perluigi, M., “Activation of p53 in Down Syndrome and in the Ts65Dn Mouse Brain is Associated with a Pro-Apoptotic Phenotype.”, J Alzheimers Dis, vol. 52, no. 1, pp. 359-71, 2016.
A. Palmeri, Mammana, L., Tropea, M. Rosaria, Gulisano, W., and Puzzo, D., “Salidroside, a Bioactive Compound of Rhodiola Rosea, Ameliorates Memory and Emotional Behavior in Adult Mice.”, J Alzheimers Dis, vol. 52, no. 1, pp. 65-75, 2016.
S. Min Son, Shin, H. - J., Byun, J., Kook, S. Young, Moon, M., Chang, Y. Jin, and Mook-Jung, I., “Metformin Facilitates Amyloid-β Generation by β- and γ-Secretases via Autophagy Activation.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1197-208, 2016.
T. Schütt, Helboe, L., Pedersen, L. Østergaar, Waldemar, G., Berendt, M., and Pedersen, J. Torleif, “Dogs with Cognitive Dysfunction as a Spontaneous Model for Early Alzheimer's Disease: A Translational Study of Neuropathological and Inflammatory Markers.”, J Alzheimers Dis, vol. 52, no. 2, pp. 433-49, 2016.
M. Ángel Ontiveros-Torres, Labra-Barrios, M. Luisa, Díaz-Cintra, S., Aguilar-Vázquez, A. Ruth, Moreno-Campuzano, S., Flores-Rodríguez, P., Luna-Herrera, C., Mena, R., Perry, G., Florán-Garduño, B., Luna-Muñoz, J., and Luna-Arias, J. Pedro, “Fibrillar Amyloid-β Accumulation Triggers an Inflammatory Mechanism Leading to Hyperphosphorylation of the Carboxyl-Terminal End of Tau Polypeptide in the Hippocampal Formation of the 3×Tg-AD Transgenic Mouse.”, J Alzheimers Dis, vol. 52, no. 1, pp. 243-69, 2016.
Q. Ye, Bai, F., and Zhang, Z., “Shared Genetic Risk Factors for Late-Life Depression and Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 1, pp. 1-15, 2016.
K. Hensley and Kursula, P., “Collapsin Response Mediator Protein-2 (CRMP2) is a Plausible Etiological Factor and Potential Therapeutic Target in Alzheimer's Disease: Comparison and Contrast with Microtubule-Associated Protein Tau.”, J Alzheimers Dis, vol. 53, no. 1, pp. 1-14, 2016.
E. Chiziane, Telemann, H., Krueger, M., Adler, J., Arnhold, J., Alia, A., and Flemmig, J., “Free Heme and Amyloid-β: A Fatal Liaison in Alzheimer's Disease.”, J Alzheimers Dis, vol. 61, no. 3, pp. 963-984, 2018.
D. E. Moss, Perez, R. G., and Kobayashi, H., “Cholinesterase Inhibitor Therapy in Alzheimer's Disease: The Limits and Tolerability of Irreversible CNS-Selective Acetylcholinesterase Inhibition in Primates.”, J Alzheimers Dis, vol. 55, no. 3, pp. 1285-1294, 2017.

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