Biblio

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H
K. Hensley and Kursula, P., “Collapsin Response Mediator Protein-2 (CRMP2) is a Plausible Etiological Factor and Potential Therapeutic Target in Alzheimer's Disease: Comparison and Contrast with Microtubule-Associated Protein Tau.”, J Alzheimers Dis, vol. 53, no. 1, pp. 1-14, 2016.
C. Henneges, Reed, C., Chen, Y. - F., Dell'Agnello, G., and Lebrec, J., “Describing the Sequence of Cognitive Decline in Alzheimer's Disease Patients: Results from an Observational Study.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1065-80, 2016.
B. C. Henley, Shokouhi, M., Mahajan, A. Y., Inan, O. T., and Hajjar, I., “Cardiovascular Response to Mental Stress in Mild Cognitive Impairment and its Association with Cerebral Perfusion.”, J Alzheimers Dis, 2018.
S. Hendriks, Peetoom, K., Tange, H., van Bokhoven, M. A., van der Flier, W. M., Bakker, C., Papma, J. M., Koopmans, R., Verhey, F., Köhler, S., and de Vugt, M., “Pre-Diagnostic Symptoms of Young-Onset Dementia in the General Practice up to Five Years Before Diagnosis.”, J Alzheimers Dis, vol. 88, no. 1, pp. 229-239, 2022.
A. Heller, Coffman, S. S., and Jarvis, K., “Potentially Pathogenic Calcium Oxalate Dihydrate and Titanium Dioxide Crystals in the Alzheimer's Disease Entorhinal Cortex.”, J Alzheimers Dis, vol. 77, no. 2, pp. 547-550, 2020.
J. Heinrich, Vidal, J. - S., Simon, A., Rigaud, A. - S., Hanon, O., Epelbaum, J., Viollet, C., and Duron, E., “Relationships Between Lower Olfaction and Brain White Matter Lesions in Elderly Subjects with Mild Cognitive Impairment.”, J Alzheimers Dis, vol. 61, no. 3, pp. 1133-1141, 2018.
M. Heffernan, Mather, K. A., Xu, J., Assareh, A. A., Kochan, N. A., Reppermund, S., Draper, B., Trollor, J. N., Sachdev, P., and Brodaty, H., “Alcohol Consumption and Incident Dementia: Evidence from the Sydney Memory and Ageing Study.”, J Alzheimers Dis, vol. 52, no. 2, pp. 529-38, 2016.
M. Heffernan, Andrews, G., Singh, M. A. Fiataron, Valenzuela, M., Anstey, K. J., Maeder, A. J., McNeil, J., Jorm, L., Lautenschlager, N. T., Sachdev, P. S., Ginige, J. A., Hobbs, M. J., Boulamatsis, C., Chau, T., Cobiac, L., Cox, K. L., Daniel, K., Flood, V. M., Guerrero, Y., Gunn, J., Jain, N., Kochan, N. A., Lampit, A., Mavros, Y., Meiklejohn, J., Noble, Y., O'Leary, F., Radd-Vagenas, S., Walton, C. C., and Brodaty, H., “Maintain Your Brain: Protocol of a 3-Year Randomized Controlled Trial of a Personalized Multi-Modal Digital Health Intervention to Prevent Cognitive Decline Among Community Dwelling 55 to 77 Year Olds.”, J Alzheimers Dis, vol. 70, no. s1, pp. S221-S237, 2019.
D. M. Hedderich, Spiro, J. E., Goldhardt, O., Kaesmacher, J., Wiestler, B., Yakushev, I., Zimmer, C., Boeckh-Behrens, T., and Grimmer, T., “Increasing Diagnostic Accuracy of Mild Cognitive Impairment due to Alzheimer's Disease by User-Independent, Web-Based Whole-Brain Volumetry.”, J Alzheimers Dis, vol. 65, no. 4, pp. 1459-1467, 2018.
M. Heath, Shellington, E., Titheridge, S., Gill, D. P., and Petrella, R. J., “A 24-Week Multi-Modality Exercise Program Improves Executive Control in Older Adults with a Self-Reported Cognitive Complaint: Evidence from the Antisaccade Task.”, J Alzheimers Dis, vol. 56, no. 1, pp. 167-183, 2017.
M. Heath, Weiler, J., Gregory, M. A., Gill, D. P., and Petrella, R. J., “A Six-Month Cognitive-Motor and Aerobic Exercise Program Improves Executive Function in Persons with an Objective Cognitive Impairment: A Pilot Investigation Using the Antisaccade Task.”, J Alzheimers Dis, vol. 54, no. 3, pp. 923-931, 2016.
D. Head, Bugg, J. M., Goate, A. M., Fagan, A. M., Mintun, M. A., Benzinger, T., Holtzman, D. M., and Morris, J. C., “Exercise Engagement as a Moderator of the Effects of APOE Genotype on Amyloid Deposition.”, Arch Neurol, vol. 69, no. 5, pp. 636-43, 2012.
J. - T. He, Zhao, X., Xu, L., and Mao, C. - Y., “Vascular Risk Factors and Alzheimer's Disease: Blood-Brain Barrier Disruption, Metabolic Syndromes, and Molecular Links.”, J Alzheimers Dis, vol. 73, no. 1, pp. 39-58, 2020.
X. - Y. He, Dobkin, C., W Brown, T., and Yang, S. - Y., “3-Hydroxyacyl-CoA and Alcohol Dehydrogenase Activities of Mitochondrial Type 10 17β-Hydroxysteroid Dehydrogenase in Neurodegeneration Study.”, J Alzheimers Dis, vol. 88, no. 4, pp. 1487-1497, 2022.
G. He, Luo, W., Li, P., Remmers, C., Netzer, W. J., Hendrick, J., Bettayeb, K., Flajolet, M., Gorelick, F., Wennogle, L. P., and Greengard, P., “Gamma-secretase activating protein is a therapeutic target for Alzheimer's disease.”, Nature, vol. 467, no. 7311, pp. 95-8, 2010.
X. - Y. He, Isaacs, C., and Yang, S. - Y., “Roles of Mitochondrial 17β-Hydroxysteroid Dehydrogenase Type 10 in Alzheimer's Disease.”, J Alzheimers Dis, vol. 62, no. 2, pp. 665-673, 2018.
C. C. Hays, Zlatar, Z. Z., Meloy, M. J., Osuna, J., Liu, T. T., Galasko, D. R., and Wierenga, C. E., “Anterior Cingulate Structure and Perfusion Is Associated with Cerebrospinal Fluid Tau Among Cognitively Normal Older Adult APOEɛ4 Carriers.”, J Alzheimers Dis, vol. 73, no. 1, pp. 87-101, 2020.
K. Hayashi, Gonzales, T. K., Kapoor, A., Ziegler, T. E., Meethal, S. Vadakkadat, and Atwood, C. S., “Development of Classification Models for the Prediction of Alzheimer's Disease Utilizing Circulating Sex Hormone Ratios.”, J Alzheimers Dis, vol. 76, no. 3, pp. 1029-1046, 2020.
J. Haworth, Phillips, M., Newson, M., Rogers, P. J., Torrens-Burton, A., and Tales, A., “Measuring Information Processing Speed in Mild Cognitive Impairment: Clinical Versus Research Dichotomy.”, J Alzheimers Dis, vol. 51, no. 1, pp. 263-75, 2016.
K. M. Hawkins and Sergio, L. E., “Adults at Increased Alzheimer's Disease Risk Display Cognitive-Motor Integration Impairment Associated with Changes in Resting-State Functional Connectivity: A Preliminary Study.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1161-72, 2016.
Y. Hattori, Maki, T., Saito, S., Yamamoto, Y., Nagatsuka, K., and Ihara, M., “Influence of Low-Dose Aspirin on Cerebral Amyloid Angiopathy in Mice.”, J Alzheimers Dis, vol. 52, no. 3, pp. 1037-45, 2016.
S. Hatashita and Wakebe, D., “Amyloid-β Deposition and Long-Term Progression in Mild Cognitive Impairment due to Alzheimer's Disease Defined with Amyloid PET Imaging.”, J Alzheimers Dis, vol. 57, no. 3, pp. 765-773, 2017.
A. Hatami, Monjazeb, S., and Glabe, C., “The Anti-Amyloid-β Monoclonal Antibody 4G8 Recognizes a Generic Sequence-Independent Epitope Associated with α-Synuclein and Islet Amyloid Polypeptide Amyloid Fibrils.”, J Alzheimers Dis, vol. 50, no. 2, pp. 517-25, 2016.
Y. Hatada, Hashimoto, M., Shiraishi, S., Ishikawa, T., Fukuhara, R., Yuki, S., Tanaka, H., Miyagawa, Y., Kitajima, M., Uetani, H., Tsunoda, N., Koyama, A., and Ikeda, M., “Cerebral Microbleeds Are Associated with Cerebral Hypoperfusion in Patients with Alzheimer's Disease.”, J Alzheimers Dis, vol. 71, no. 1, pp. 273-280, 2019.
M. Hashimoto, Ho, G., Sugama, S., Takamatsu, Y., Shimizu, Y., Takenouchi, T., Waragai, M., and Masliah, E., “Evolvability of Amyloidogenic Proteins in Human Brain.”, J Alzheimers Dis, vol. 62, no. 1, pp. 73-83, 2018.

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