Biblio
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“Different Expression Patterns of Amyloid-β Protein Precursor Secretases in Human and Mouse Hippocampal Neurons: A Potential Contribution to Species Differences in Neuronal Susceptibility to Amyloid-β Pathogenesis.”, J Alzheimers Dis, vol. 51, no. 1, pp. 179-95, 2016.
, “Different Patterns of Correlation between Grey and White Matter Integrity Account for Behavioral and Psychological Symptoms in Alzheimer's Disease.”, J Alzheimers Dis, vol. 50, no. 2, pp. 591-604, 2016.
, “Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
, “Differential Diagnosis of Dementia with High Levels of Cerebrospinal Fluid Tau Protein.”, J Alzheimers Dis, vol. 51, no. 3, pp. 905-13, 2016.
, “Differential Mass Spectrometry Profiles of Tau Protein in the Cerebrospinal Fluid of Patients with Alzheimer's Disease, Progressive Supranuclear Palsy, and Dementia with Lewy Bodies.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1033-43, 2016.
, “The Disconnection Hypothesis in Alzheimer's Disease Studied Through Multimodal Magnetic Resonance Imaging: Structural, Perfusion, and Diffusion Tensor Imaging.”, J Alzheimers Dis, vol. 50, no. 4, pp. 1051-64, 2016.
, “Discovery and Subsequent Confirmation of Novel Serum Biomarkers Diagnosing Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 2, pp. 317-27, 2016.
, “Discrepancy in Expression of β-Secretase and Amyloid-β Protein Precursor in Alzheimer-Related Genes in the Rat Medial Temporal Lobe Cortex Following Transient Global Brain Ischemia.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1023-31, 2016.
, “Diversity of Cognitive Phenotypes Associated with C9ORF72 Hexanucleotide Expansion.”, J Alzheimers Dis, vol. 52, no. 1, pp. 25-31, 2016.
, “Diversity of Cognitive Phenotypes Associated with C9ORF72 Hexanucleotide Expansion.”, J Alzheimers Dis, vol. 52, no. 1, pp. 25-31, 2016.
, “Diversity of Cognitive Phenotypes Associated with C9ORF72 Hexanucleotide Expansion.”, J Alzheimers Dis, vol. 52, no. 1, pp. 25-31, 2016.
, “Drawing Disorders in Alzheimer's Disease and Other Forms of Dementia.”, J Alzheimers Dis, vol. 53, no. 1, pp. 31-52, 2016.
, “Dual Kinase Inhibition Affords Extended in vitro Neuroprotection in Amyloid-β Toxicity.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1659-1670, 2016.
, “Dysregulation of Autophagy, Mitophagy, and Apoptotic Genes in the Medial Temporal Lobe Cortex in an Ischemic Model of Alzheimer's Disease.”, J Alzheimers Dis, vol. 54, no. 1, pp. 113-21, 2016.
, “Dysregulation of Elongation Factor 1A Expression is Correlated with Synaptic Plasticity Impairments in Alzheimer's Disease.”, J Alzheimers Dis, vol. 54, no. 2, pp. 669-78, 2016.
, “Early Postmenopausal Transdermal 17β-Estradiol Therapy and Amyloid-β Deposition.”, J Alzheimers Dis, vol. 53, no. 2, pp. 547-56, 2016.
, “EEG Markers of Dementia with Lewy Bodies: A Multicenter Cohort Study.”, J Alzheimers Dis, vol. 54, no. 4, pp. 1649-1657, 2016.
, “Effect of Normal Aging and of Mild Cognitive Impairment on Event-Related Potentials to a Stroop Color-Word Task.”, J Alzheimers Dis, vol. 52, no. 4, pp. 1487-501, 2016.
, “The Effects of Latrepirdine on Amyloid-β Aggregation and Toxicity.”, J Alzheimers Dis, vol. 50, no. 3, pp. 895-905, 2016.
, “Efficacy and Safety of ABT-126 in Subjects with Mild-to-Moderate Alzheimer's Disease on Stable Doses of Acetylcholinesterase Inhibitors: A Randomized, Double-Blind, Placebo-Controlled Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1237-47, 2016.
, “Efficacy and Safety of ABT-126 in Subjects with Mild-to-Moderate Alzheimer's Disease on Stable Doses of Acetylcholinesterase Inhibitors: A Randomized, Double-Blind, Placebo-Controlled Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 1237-47, 2016.
, “Efficacy and Safety of Crocus sativus L. in Patients with Mild Cognitive Impairment: One Year Single-Blind Randomized, with Parallel Groups, Clinical Trial.”, J Alzheimers Dis, vol. 54, no. 1, pp. 129-33, 2016.
, “Evaluation of a Genetic Risk Score to Improve Risk Prediction for Alzheimer's Disease.”, J Alzheimers Dis, vol. 53, no. 3, pp. 921-32, 2016.
, “Expression of Tau Produces Aberrant Plasma Membrane Blebbing in Glial Cells Through RhoA-ROCK-Dependent F-Actin Remodeling.”, J Alzheimers Dis, vol. 52, no. 2, pp. 463-82, 2016.
, “Extracellular Vesicles Containing P301L Mutant Tau Accelerate Pathological Tau Phosphorylation and Oligomer Formation but Do Not Seed Mature Neurofibrillary Tangles in ALZ17 Mice.”, J Alzheimers Dis, vol. 54, no. 3, pp. 1207-1217, 2016.
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