Biblio
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“Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Changes in Functional Brain Connectivity Predicts Conversion to Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 2, pp. 377-89, 2016.
, “Longitudinal Memory Profiles in Behavioral-Variant Frontotemporal Dementia and Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 775-82, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Longitudinal Protein Changes in Blood Plasma Associated with the Rate of Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 4, pp. 1105-14, 2016.
, “Low Plasma Cholinesterase Activities are Associated with Deficits in Spatial Orientation, Reduced Ability to Perform Basic Activities of Daily Living, and Low Body Mass Index in Patients with Progressed Alzheimer's Disease.”, J Alzheimers Dis, vol. 51, no. 3, pp. 801-13, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Prevalence of Mixed Dementia in a Cohort of 2,000 Elderly Patients in a Memory Clinic Setting.”, J Alzheimers Dis, vol. 50, no. 3, pp. 797-806, 2016.
, “Low Serum Insulin-Like Growth Factor-I Predicts Cognitive Decline in Alzheimer's Disease.”, J Alzheimers Dis, vol. 52, no. 2, pp. 641-9, 2016.
, “Lower Late-Life Body-Mass Index is Associated with Higher Cortical Amyloid Burden in Clinically Normal Elderly.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1097-105, 2016.
, “Lower Late-Life Body-Mass Index is Associated with Higher Cortical Amyloid Burden in Clinically Normal Elderly.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1097-105, 2016.
, “Lower Prevalence of Alzheimer's Disease among Tibetans: Association with Religious and Genetic Factors.”, J Alzheimers Dis, vol. 50, no. 3, pp. 659-67, 2016.
, “LRP/LR Antibody Mediated Rescuing of Amyloid-β-Induced Cytotoxicity is Dependent on PrPc in Alzheimer's Disease.”, J Alzheimers Dis, vol. 49, no. 3, pp. 645-57, 2016.
, “Maintenance of Cognitive Performance and Mood for Individuals with Alzheimer's Disease Following Consumption of a Nutraceutical Formulation: A One-Year, Open-Label Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 991-5, 2016.
, “Maintenance of Cognitive Performance and Mood for Individuals with Alzheimer's Disease Following Consumption of a Nutraceutical Formulation: A One-Year, Open-Label Study.”, J Alzheimers Dis, vol. 51, no. 4, pp. 991-5, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “MAPT H1 Haplotype is Associated with Late-Onset Alzheimer's Disease Risk in APOEɛ4 Noncarriers: Results from the Dementia Genetics Spanish Consortium.”, J Alzheimers Dis, vol. 49, no. 2, pp. 343-52, 2016.
, “Measurement Error, Reliability, and Minimum Detectable Change in the Mini-Mental State Examination, Montreal Cognitive Assessment, and Color Trails Test among Community Living Middle-Aged and Older Adults.”, J Alzheimers Dis, vol. 53, no. 3, pp. 1107-14, 2016.
, “The Mechanism Underlying Amyloid Polymorphism is Opened for Alzheimer's Disease Amyloid-β Peptide.”, J Alzheimers Dis, vol. 54, no. 2, pp. 821-30, 2016.
, “The Mechanism Underlying Amyloid Polymorphism is Opened for Alzheimer's Disease Amyloid-β Peptide.”, J Alzheimers Dis, vol. 54, no. 2, pp. 821-30, 2016.
, “The Mechanism Underlying Amyloid Polymorphism is Opened for Alzheimer's Disease Amyloid-β Peptide.”, J Alzheimers Dis, vol. 54, no. 2, pp. 821-30, 2016.
, “The Memory Binding Test: Development of Two Alternate Forms into Spanish and Catalan.”, J Alzheimers Dis, vol. 52, no. 1, pp. 283-93, 2016.
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